Gluten OR FODMAP?

Gluten OR FODMAP?

Sometimes people go Gluten free when they have irritable bowel issues. If however, when symptoms do not resolve, having some formal testing helps. Integrative Medicine Doctors generally test for Coeliac Haplotype. Having a Haplotypes of DQ2 or DQ8 increases the risk not only to gluten but other autoimmune issues.

Sometimes, ruling out wheat and other Gluten products, and the issues of abdominal irritable bowels such as bloating and recurrent gut pain persists, the cause of these symptoms may be due to poorly absorbed sugars collectively known as FODMAPs. 

Wheat happens to be high in certain FODMAPs.  Peter Gibson of The Alfred Hospital and Monash University in Melbourne, Australia says that we can learn and figure out which foods affect our gut and can adjust our diet according to symptoms. 

FODMAPS stands for:

Fermentable

The process through which gut bacteria degrade undigested carbohydrate to

 produce gases (hydrogen, methane and carbon dioxide)

Oligo-saccharides

1.  Fructo-oligosaccharides (FOS) found in; wheat, rye, onions and garlic

2.  Galacto-oligosaccharides (GOS) found in ; legumes/pulses

Disaccharides

Lactose found in; milk, soft cheese, yoghurts

Mono-saccharide

Fructose (in excess of glucose) found in honey,

 apples, high fructose corn syrups

Polyols

 Sugar polyols (eg. sorbitol, mannitol) found in some fruit and vegetables and

 used as artificial sweeteners

http://www.med.monash.edu/cecs/gastro/fodmap/description.html

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Health

John Skelton – Newsletter Editor Retires

It is with sadness that John has decided to step down from being the Editor of our newsletter because of ill health.

We all owe a huge debt of gratitude to John because if he had not attended lectures by Dr Abram Hoffer and Dr Carl Pfeiffer in Sydney in the 1980’s and felt that these ideas must be introduced to Australia, Bio-Balance may never have been established.

As a caring father John, together with his wife Margaret tried everything possible to help their daughter Julie recover from Schizophrenia and are still doing so today.   In order to find ways to help her John  travelled overseas to try to find help and whilst in Canada heard Dr Bill Walsh lecture at the Nutrition Today Conference and were impressed by his scientific approach.

Dr Walsh kindly agreed to come to Australia to train a doctor on the Gold Coast, Richard Stuckey.  John was very involved in the establishment of the Bio-Balance Health Association as a not-for-profit entity and helped organise the first training.  Shortly thereafter he transferred to Eden in NSW to be closer to his son, but always kept in close contact by being the Editor of our Newsletter and played a very valuable role as Vice President for many years.

John was appointed a Member of the Order of Australia for his services to mental health – he had many other roles in Queensland as well as his work for Bio-Balance – a very well deserved reward for many, many years of volunteer work to help those with a mental illness.

We wish him well and will always keep him in the loop of the progress of the organization.

Thank you John.

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General

The Walsh Theory Of Schizophrenia

A Novel Theory Of Schizophrenia

A flaw in existing theories has been the failure to recognize that schizophrenia is an umbrella term that includes at least three very different disorders, each presenting a distinctive set of symptoms and traits. It seems most unlikely that these disparate mental illnesses arise from the same underlying cause. I believe a proper theory of schizophrenia must include the following elements:

  • Separate causation for the major phenotypes,
  • Explanation for the “mental-breakdown” event that usually occurs in late adolescence or young adulthood,
  • Explanation for the life-long persistence of schizophrenia after the mental breakdown, and
  • Explanation why this familial (heritable) disorder violates classical laws of Mendelian genetics.

Common features of the different types of schizophrenia include (a) relative normalcy prior to the mental breakdown, (b) psychosis, (c) cognitive deficits, (d) loss of social skills, (e) high anxiety, (f) enlarged brain ventricles and smaller volumes in cortex and other brain areas, and (g) benefits often achieved using atypical antipsychotic medications. There is a common thread in these disorders, despite their very great differences in biochemistry. Eventually I realized that the three major schizophrenia phenotypes shared the following important features: (1) vulnerability to epigenetic errors that can alter gene expression, and (2) weakened protection against oxidative stress. These insights led to my theory of schizophrenia which is presented below:

Walsh Theory Of Schizophrenia

Thesis 1:  Schizophrenia is Epigenetic in Nature:  A psychotic “breakdown” is usually followed by a lifetime of mental illness and misery. This often permanent change in functioning results from altered chromatin bookmarks that regulate gene expression. Since the deviant marks are maintained during future cell divisions, the condition doesn’t “go away”.

Thesis 2: Weak Antioxidant Protection is a Distinctive Feature of Schizophrenia:  Most schizophrenics exhibit a genetic or acquired weakness in antioxidant protection. Evidence from my extensive chemistry database includes generally low levels of glutathione, cysteine, selenium, zinc, polyunsaturated fats, together with high levels of non-ceruloplasmin copper. 

Thesis 3:  Oxidative Overload Produces Deviant Epigenetic Marks in Schizophrenia: Cancer researchers have identified cumulative oxidative stress as a trigger that can transform healthy cells into cancer cells by altering epigenetic marks that permanently change gene expression. Examples include (a) skin cancer developing after years of excessive sun exposure, and (b) lung cancer following years of cigarette smoking. It’s not a coincidence that nearly all schizophrenia patients exhibit excess oxidative stress. The onset of schizophrenia occurs when oxidative stresses exceed the threshold level needed to alter chromatin marks that regulate gene expression.

Thesis 4:  Methylation Imbalances Promote Epigenetic Vulnerability to Oxidative Stress:  Abnormal methylation of chromatin is a leading cause of epigenetic errors in gene expression. The combination of oxidative overload and a methyl imbalance can produce gene expression changes that result in a chronic schizophrenia condition. The two most prevalent forms of schizophrenia develop in persons who exhibit either (a) methyl overload or (b) methyl deficiency. The two resulting psychotic disorders exhibit very different brain chemistry and symptoms.

A. Overmethylation – About 46% of persons diagnosed with schizophrenia exhibit excessive methylation of chromatin along with weak antioxidant protection. Mental breakdowns generally occur during severe physical or emotional traumatic events that produce overwhelming oxidative stress and deviant gene marks. This schizophrenia biotype is a sensory disorder that generally involves auditory, tactile, or visual hallucinations. This condition is associated with elevated activity of dopamine and norepinephrine, and reduced glutamate activity at NMDA receptors.  The most common DSM-4 diagnosis is paranoid schizophrenia.

B. Undermethylation – About 28% of persons diagnosed with schizophrenia exhibit low methylation of chromatin together with weak antioxidant protection. Mental breakdowns generally occur during severe physical or emotional traumatic events that produce a separate set of altered gene marks. This schizophrenia biotype essentially is a thought disorder with delusions and catatonic tendencies the primary symptoms. This condition is associated with low activity at serotonin, dopamine, and NMDA receptors. The most common DSM-4 diagnoses are Schizoaffective Disorder or Delusional Disorder.

Thesis 5: Extraordinary Weakness in Antioxidant Protection Can Produce Schizophrenia in the Absence of Methyl Imbalances:  The third major schizophrenia phenotype develops in persons with an inborn severe deficit in antioxidant protection. This condition is arbitrarily termed “Pyrrole Disorder” due to the presence of excessive pyrrole levels in blood and urine. Mental breakdowns occur for these persons during periods of extreme physical or mental stress in which deviant epigenetic marks are established. This condition is characterized by extraordinary anxiety, rapid mood swings, and often involves both auditory hallucinations and delusional beliefs. Brain chemistry abnormalities include (a) depressed glutamate activity at NMDA receptors, and (b) very depressed GABA activity.

Thesis 6:  Failure to Follow Classical Laws of Genetic Inheritance Results From the Epigenetic Nature of Schizophrenia:  Schizophrenia is strongly heritable (runs in families) but fails to obey Mendel’s classic laws of genetic inheritance. There are countless examples of identical twins where one sibling develops the disorder and the other does not. In addition, intensive research efforts to identify the schizophrenia gene (or genes) have met with little success. Epigenetics provides two explanations for the non-Mendelian nature of schizophrenia: (a) Environmental insults are required to produce deviant epigenetic marks and environmental conditions are highly variable for different individuals, and (b) Transgenerational epigenetic inheritance (TEI) contributes to schizophrenia heritability by transmitting deviant epigenetic marks to one’s children and grandchildren.

Purchase Nutrient Power by William J Walsh from The Walsh Research Institute

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Books | Schizophrenia

Summary of Dr. Julie Buckley’s Presentation – Outreach Conference 2014

Dr Buckley delivered a very in-depth talk about Autism and the gut. Dr Buckley stated that we have to unlearn things we have learnt to accommodate new research and presented a Functional Model of Autism.

The lecture, in point form is summarized as follows:

  • The interaction of communication problems, gut disease and toxins overlapping
  • socialisation problems, immune dysregulation, chronic inflammation
  • behavioural problems, methylation interruption and oxidative stress. 

Dr Buckley said that if we manage the underlying biochemistry then the  behavioural issues will take care of themselves. 

Dr Buckley spent a significant amount of time on gut inflammation and Autism. Endoscopic evidence of the gut inflammation which looks like Crohns but is not Crohns was presented. These Autistic traits appear to present as behavioural  problems  in children when  they cannot talk.
Dr Buckley introduced THE FOUR Rs : 

  • Remove  - offending foods, EMF exposures, unnecessary toxic exposures.  
  • Repair – heal the leaky bowel, dietary approaches – GFCG/GAPS/SCD.Paeo/ grain Free/Ketogenic, 
  • Replace – probiotics, Digestive enzymes with DPPIV, 
  • Restore – nourish the body – often at replacement values, Exercise – the bowel is designed to move.

Dr Buckley pointed out that:

Bowel symptoms warrant endoscopies 

  • especially with sudden and unexpected behavioural change - hallmark of GIT issues
  • The pathology in bowel disease in ASD is largely inflammatory in nature
  • It occurs along the entire length of the bowel form, mouth to anus
  • Constipation an issue in ASD kids
  • Apthous ulcers & white spot lesions in histology on scopes
  • Oesophinolic oesophagitis, sometimes not reported in the report
  • Erosive ulcer: sees protein loss – absorption an issue

Dr Buckley presented  a paper on -  Hypothesis of etiology of IBD in ASD (Ref: Strober W, The fundamental basis of IBD. J.Clin. Invest. 117. 514 -521 (2007)

  • Abnormal  dysregulated immune system, normal gut microbes OR
  • Normal immune system, abn microbes +/- abn barrier  
  • Conclusion : That IBD is characterized by an abnormal mucosal immune response but that microbial factors & epithelial cell abn can facilitate this response

Dr Buckley also spoke about talked behaviours in Autistic kids that point towards Inflammatory Bowel Disease( IBD )

  • standing on tip toes
  • doubling over
  • unsettled crying in the middle of the night

Dr Buckley then went on to talk about Dietary approaches and presented studies which show that Gluten free, Casein free diets do make a difference. 

Ref:  Pennesi, Christine M; Klein, Laura Cousino. Nutritional Neuroscience, Vol 15, No2, March 2012, pp 85-7 (-77). Maney Publishing. Effectiveness of the gluten-free, casein-free diet for children diagnosed with autism spectrum disorder: Based on parental report.   

  • Diet CFCF diet Must be healthy
  • As organic as possible, especially the “dirty dozen”
  • Focused on nuts, seeds, healthy fats, and veggies – ketogenic or modified Atkins approach
  • Carb minimized
  • Paleo/Grain free/SCD
  • Veggies is the best source of calcium in leafy green veggies
  • www.tacanow.org : great section on GFCF diet
  • Many kids experience sig opiate withdrawal symptoms – prepare to manage these with Epsom salts baths and / or deactivated charcoal
  • Ketogenic diets are good for kids with seizures

Dr Buckley emphasised the critical importance of healthy fats 

  • medium chain fatty acids ( coconuts, nut oils, palm kernel oil)
  • MCT enter cells without carnitine assistance: more efficiently and bountiful ATP production by mitochondria
  • Diets higher in healthy fat = Less carb seeking, More willing diversification of diet, Easier control of yeast overgrowth [Mitochondrial dysfunction see often in Autism] and eating your colours.

In referring to the R’s -  Repair and restore: Vitamins and minerals

Augment relative deficiencies - Daily values are not what is necessary, Daily values are what is needed to prevent a deficiency state disease, Vitamins and minerals are co-factors to most enzymatic reactions in the body 

  • Vitamins
  • Vit A,D, E & Vit C,B6,B-12 and appropriate form of folate
  • Minerals  - Se, Zn, Zn, Fe, Cu

Dr Buckley stressed that we have to refill dry wells in these kids – that they have had years of chronic malabsorption and so supplementation may take years before they have enough stores to come off supplements. Dr Buckley talked about:

  • the bowel is not inside us
  • it is a tube that runs thru & is the outside world
  • Gut-immune interface 

Dr Buckley went on to talk about PROBIOTICS

  • critical to digestion
  • significant impact neurotransmitter function
  • and is the foundation of immune competence

In Talking about IBD, she presented research on Dysbiosis

  • an imbalance in gut flora
  • should not see clostridium – propionic acid can precipitate symptoms of autism (MacFae, D; Behav Brain res. 2007. Jan 10; 176 (1) 146 – 69)
  • the norm – may not be normal. More bacteria than our cells in our body
  • more Metabolic activity than in liver 

Ref: Bacteroides fragilis corrects gut permeability, alters microbial composition, and ameliorates degects in communicative stereotype, anxiety-like and sensorimotor behaviours ( in mouse models mimicking autism)
Hsioa, E. et al
Microbiotita Modulate behavioural and physiological abnormalities Asc with neurodevelopmental Disorders.
Cell 155, 1451 – 1463, Dec 2013
Confirms probitocs impact neurotransmitter function

In talking about the management of IBD

  • Dr Buckley said “if we can change the flora, we can change the autism”
  • must ensure the diet is altered to support and sustain the new regimen of flora once they are introduced
  • changing the flora without changing the enviroment in which they will be living is like putting fresh water for salt water fish

Resetting the bowel

  • eradicate what’s there (Antimicrobials, Anti parasitic, Antifungals )
  • Be prepared to start with a lower dose and increase rapidly
  • work the biofilm first if truly needed 
  • Continue regular dosing of probiotics

Resetting the bowel – Probiotic transplantation

  • diverse sources help to ensure larger sample of DNA for the immune system to monitor

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From The President July 2014

Our 11th Medical Practitioner Training and Patient assessments began on 5th April, 2014 at the Outrigger Resort Hotel at Surfers Paradise.

We were delighted that we had 64 doctors attend with 30 for the first time and 4 psychiatrists also attended.   Training was given by Dr Bill Walsh, Dr Albert Mensah and Dr Judith Bowman, with lectures from Dr Liz Mumper, Dr John Criticos, Dr Kerry Harris and Dr Henry Butt .  A number of doctors said that it exceeded their expectations (which is always a good thing to do) and one said she now felt she was a “proper” doctor and could help many in her practice from what she had learnt during the week.

The Conference was a great success with 20 stands, and over 300 attendees.  Speakers in the morning beginning at 10 am were Dr Liz Mumper, Dr Julie Buckley and Dr Lily Tomas.  A lively Q & A session followed.

In the afternoon, we had a report on a new research project which is being undertaken at Griffith University on children’s aggressive behavior, then Dr Jerome Sarris from the International Network of Integrative Mental Health and University of Melbourne spoke followed by Dr William Walsh.   Lastly we had  a Q & A session .  We received very good feedback about the conference day.

We had 160 patients attend during the week, and we have received some very good feedback from them as well, so we do hope they have all found it a rewarding experience.

Our next Outreach will be 21- 28th March 2015 at the same hotel in Surfers Paradise.

We have to thank so many people for their help and assistance during the Conference and the whole week – we found some wonderful new volunteers so we are extremely grateful for that.   Angela Testa for her work with the Doctors Manuals,  Margaret Harms who worked under some difficulty during this time,  Marnie Lo, Bruce Jeanes.   We have also had offers from some people to run small seminars to raise awareness and we feel that would be a wonderful idea and if we can be of assistance please let us know.

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General | Outreach

Outreach Report 2014

The 2014 Gold Coast Outreach Patient Assessment bookings are currently full steam ahead for another exciting year. Assessments are filling up fast. We are already over half way filled with many patients taking advantage of the Early Bird Special that ended December 5th. This is the time of year where many parents are enjoying their lovely children at home during the school holiday period -- a fantastic opportunity to see them up close and personal while carefully monitoring their Biomedical progress. It is usually towards the end of January that we have a second wave of parents signing up for another Outreach hoping to take their child’s treatment protocols to the next level. If you feel you are one of these parents, I encourage you to help us meet your goals by booking in as soon as possible to avoid any disappointment with appointment availability.

Similarly, thanks to our doctors and friends at ACNEM, we continue to get a steady stream of new practitioners signing up to train with us. So far we are thrilled to have on board 13 new doctors, our goal being 20. We welcome these doctors warmly to our training program and we are excited about this sudden new wave of integrative medical acceptance and willingness to learn. The future ahead for our patients looks very exciting indeed! 

To help mentor and support our new doctors, we currently have 24 previously trained doctors enrolled returning to update their skills, and expect at least another 10, thereby signing up approximately 47 with many more sneaking in at the last minute. 

As Co-ordinator, I am always amazed at the number of doctors we have attending each Outreach- not only signing up for the first time but also returning year after year, which is testament to the high calibre quality of our training course, therefore making all of us at Bio Balance extremely proud!

We currently have a few weeks to go until the next Outreach. As a patient, you would need to quickly book in with your Bio-Balance trained doctor, organise testing and also book in with me. Time flies, so it is best to organise this as soon as possible

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General | Outreach | Outreach Conference

Potential Financial Savings By Using Targeted Nutritional Treatment Of Mental Health

Dr Richard Stuckey

One Schizophrenia patient who was in a floridly psychotic phase at the time of consultation. For 3 years prior to his appointment he had spent an average of 30 days per year in hospital.

Over the next decade he has lived independently. He exercises, shops, catches public transport, is in regular contact with his psychiatrist, takes both pharmaceutical and nutritional medicine, is not employed and has not been in hospital.

If his prior hospital admission rate had persisted he would have had 300 hospital days over 10 years and al-lowing $1500 a day this would have amounted to $450,000. A considerable saving to Medicare for only one patient.

In 2010 I published a 12 month follow up on 567 consec-utive patients who consulted me . Of the 157 patients with a diagnosis of depression, Bipolar, Addictive Disorder and Schizophrenia they had accumulated approx 300 hospital admission days.

157 treated patients , 300 hospital days - 2 hospital days per patient.

We were able to contact 26 patients who did not start this program - between them they had accumulated 650 hospital days in one year.

26 untreated patients, 650 hospital days, 25 hospital days per patient.

If the treated group had not attended and had the same hospital rate as the non treated group there would have been 3635 extra hospital days and allowing $1500 per day there was an estimated saving to Medicare of $5,435,500 by 1 doctor in 1 year.

These comments were made at the 2013 Anniversary Dinner.

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Schizophrenia

Epigenetics 101

by William J. Walsh, Ph.D.
Walsh Research Institute
May 2013

Epigenetics is the system that determines gene regulation in humans. We have more than 20,000 genes and the only function of each gene is to make a specific protein. If the protein promotes a chemical reaction, it is called an enzyme. Every cell in the body contains an identical copy of DNA, but every tissue requires a unique combination of proteins and enzymes for optimal health. This selective production of proteins is achieved in early fetal development by a remarkable process called “bookmarking.”  For DNA, the marks involve the presence or absence of methyl groups at certain locations along the double helix. In general, methylation tends to inhibit or prevent gene expression, and the absence of a methyl mark tends to promote expression. Once established in the womb, the marks are firmly in place and usually persist throughout life. Environmental insults can produce deviant marks in the womb or later in life, and this is the cause of many physical and mental disorders.

In addition to direct methylation of DNA, gene expression of proteins may be controlled by chemicals that attach to histone proteins that provide the support structure for DNA. Histones are made up of eight linear proteins that are twisted together like a ball of yarn. DNA gently wraps around the histone balls due to electrostatic attraction: DNA is a weak acid and histones are slightly basic. In the case of histones, gene expression is often controlled by a competition between methyl and acetyl groups at histone “tails” that protrude from the ball configuration. If methyl wins the war, gene expression is inhibited. If acetyl dominates, expression of the protein is promoted.

Expression of a gene can occur only if certain large molecules can access the gene and its promoter region. Molecules called RNA polymerase are constantly swimming in the cell’s fluids looking for an exposed gene to express. Together with large molecules called transcription factors, RNA polymerase can produce a copy of the gene (called messenger RNA) that can escape through tiny pores in the cell’s nucleus and travel to the cell’s protein-production area (the reticulum). Methylation increases the basic charge of a histone whereas acetyl groups make histones less basic. By this mechanism, acetyl groups reduce the attraction between DNA and its histone, causing the DNA to uncoil from the histone and be available for expression. Methylation has the opposite effect causing the DNA and histone to compress and inhibit expression.

Most epigenetics research has been directed toward cancer and heart disease, but it’s becoming increasingly clear that many mental disorders are epigenetic in nature. The list includes autism, bipolar disorder, paranoid schizophrenia, schizoaffective disorder, post-traumatic stress, OCD, and antisocial-personality disorder. In most cases, an epigenetic disorder appears quite suddenly after a lifetime of relative wellness. Since these disorders involve deviant marks that survive cell divisions, the condition doesn’t “go away” and can persist for the remainder of life. Examples are regressive autism, Wilson’s Disease, and the sudden mental breakdowns often observed in bipolar and schizophrenia disorders.

It appears these conditions usually are caused by sudden or cumulative environmental insults in persons with a tendency for high oxidative stress. The environmental triggers may involve physical injury, illness, toxic metals, powerful medications, or emotional trauma. A gradually worsening environment may be responsible for recent epidemics of autism, breast cancer, and many other disorders.

The good news is that the gene regulation abnormalities from deviant marks appear to be reversible, suggesting a potential cure for epigenetic disorders. I can imagine a future in which newborns are scanned for deviant bookmarks, followed by treatment to normalize these chemical tags. This could eliminate the predispositions for cancer, heart disease and mental disorders that have plagued society for centuries. 

Although the technology for reversing deviant bookmarks is still unavailable, effective therapies for treatment of many epigenetic disorders are known today. For example, many paranoid schizophrenics exhibit excessive dopamine activity that can be normalized by Vitamin B-3 that uncoils DNA to increase gene expression of DAT proteins. In another example, methionine and SAMe act as serotonin reuptake inhibitors by compacting chromatin to reduce production of SERT transport proteins. We don’t yet know how to reverse deviant marks, but epigenetic science is guiding the development of therapies that uncoil or compact DNA to counter abnormal gene expression. It appears that nutrients and other natural substances are especially promising for dealing with epigenetic disorders. I believe the need for psychiatric medications will gradually fade away as science advances.

About the author:  Dr. William J. Walsh is president or the nonprofit Walsh Research Institute in Illinois and directs physician-training programs in Australia, Ireland, Norway and other countries. Dr. Walsh has authored more than 200 scientific articles and reports and has five patents. He has presented his experimental research at the American Psychiatric Association, the U.S. Senate, the National Institute of Mental Health, and has been a speaker at more than 30 international conferences. He has developed biochemical treatments for patients with behavioral disorders, ADHD, autism, depression, anxiety disorders, schizophrenia, and Alzheimer’s disease that are used by doctors throughout the world.

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Autism | Bipolar Disorder | Schizophrenia

From The President August 2013

Our 10th Anniversary Medical Practitioner Training and Patient Assessments began on 6th April, 2013 at a new venue, the Outrigger Resort Hotel, in a new city, Surfers Paradise.

We were delighted that 52 doctors registered to attend, with 20 new doctors attending for the first time. Training was given by Drs Albert Mensah and Judith Bowman with additional lectures by Dr Liz Mumper, Dr Julie Buckley, Dr Annabel Stuckey, Dr Frank Golik, Dr Kerry Harris and Dr Richard Stuckey. It was great to see how much the doc-tors who had attended in previous years looked forward to meeting up with the group again and how warmly they welcomed the newcomers.

The Conference on the 7th April attracted 270 attendees, who appreciated the lectures from our USA Faculty as well as Dr Woody McGinnis from USA/NZ and Dr Kerry Harris from South Australia. The Q &A sessions were very popular with the public as people were able to query all the doctors who had lectured. People were also milling around the 10 stallholders who were very happy with the response from the public. Amongst the stalls, we were selling Bill Walsh’s and Julie Buckley’s books. As well, some young Mums had developed business to help those families with Autistic children and we were happy to give them a platform to show their products. The whole day had a great buzz.

For our 10th Anniversary, we celebrated with a dinner in the evening, which was well attended by many of our doctors, people who have been our supporters over the years, and some of our sponsors. As well, some of the Universities also supported us by taking a table each. The theme of the evening was black and white and most people got into the spirit of the night by dressing appro-priately. A surprise of the evening was the “Singer Wait-ers” who entertained us with a selection of light Opera songs and we all joined in singing these lovely songs.

We had 175 patients attend. Some are apprehensive in not quite knowing what to expect, but in every case when they leave they are all feeling positive and hopeful. Many who came from Dr Buckley’s room could not wait to get started on a new protocol and it was helped by the fact that she had recovered her own child and was very practical in what the families could do and achieve. Some loved the fact that Dr Mumper gave them a very orga-nized protocol to follow and of course, everyone loved Dr Walsh’s approach. Their Australian doctors also learnt a great deal in these sessions.

Since we received many requests to have it at the same venue and the same place, we have already booked the space for 2014 from the 5th April to the 12th and this time we hope to provide some really lovely weather for our interstate and overseas visitors as well - unlike the weather this year.

I would like to thank all those who worked so hard and contributed so much to the success of this Event: Marnie Lo, Margaret Harms, Angela Testa, Bruce Jeanes and the many other people who are helping to spread the word around as well as all the generous help we received from people on the Conference Day itself. We very much ap-preciate it.

Judy Nicol

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General | Outreach | Outreach Conference

A Proposal for Prevention of School Shootings

William J. Walsh, PhD
Robert A. deVito, MD
January 10, 2013

Background

There have been more than 40 school shootings in the USA since 1990. If no effective action is taken, many more innocent children will die in the next few years. In the wake of the Newtown, Connecticut massacre of 20 children and six adults, the two most frequently offered solutions are (a) getting rid of the guns, and (b) keeping guns away from mentally-challenged persons. Although well-intentioned, both of these solutions would take many years to become effective.

Gun Control:  There are more than 300 million guns in the USA and more than a third of all families have a gun in the house. Effectively reducing the number of guns in the USA would require many years, even if lawmakers could agree to limit access to guns. Given today’s opposition to gun control in Congress, such laws are unlikely in the near-term future. 

Keeping guns away from mentally-challenged persons:  There are millions of children and adults in the USA who develop anxiety and depression. Some of these persons have owned guns prior to development of their mental problems. Many more live in a household with guns belonging to others. Although a significant percentage of depressives become suicidal, very few are motivated to visit schools and murder children. It would be impossible to identify the rare individuals who have this inclination.

Conclusion:  Neither of the above two “solutions” could effectively prevent school shootings for several years. In the meantime, many more children and educators will die.

A Proposal for Rapid and Efficient Prevention of School Shootings

Examination of prior school shootings reveals that most of the offenders did not exhibit violent behaviors during their formative years, and many were excellent students. Their problems developed after treatment with antidepressants or other powerful medications (see www.ssristories.com/index.php?p=school). 

These drug medications have helped millions of persons, but psychiatrists have known for years that a rare side effect involves development of suicidal ideation and in some cases homicidal tendencies. For example, SSRI antidepressants obtained from pharmacies contain an insert that warns of “possible development of suicidal ideation”. There is considerable published literature that indicates young males are especially at risk for this side effect.

Mainstream psychiatry’s “treatment of choice” for depression is use of SSRI (Selective Serotonin Reuptake Inhibitor) antidepressants aimed at increasing activity of a neurotransmitter called serotonin in the brain, perhaps coupled with counseling. However, my biochemical studies of 3,600 depression patients show that 18% have elevated serotonin activity and are intolerant to SSRI antidepressants. In most cases these persons stop compliance after discovering that the drug makes their condition worse. In many unfortunate cases, however, the patient obeys their doctor’s orders and the result is suicide.

Another danger of antidepressant drugs is sudden non-compliance. Long-term use of SSRI drugs alters the population of serotonin receptors by a mechanism called “down-regulation”.  Persons who suddenly stop an SSRI antidepressant may experience severe drug-withdrawal and become suicidal. There are several cases of school shootings in which the crime occurred soon after the offender stopped his medication.

Unique Biochemistry of Persons Intolerant to SSRI Antidepressants: More than 90% of depressives who experience symptom worsening after drug therapy exhibit a combination of high methyl and low folate levels in blood. Inexpensive laboratory testing can identify these persons who must avoid SSRI antidepressants. These persons respond better to benzodiazepine medications (e.g., Klonopin, Xanax -- see list of benzodiazepine drugs at en.wikipedia.org/wiki/List_of_benzodiazepines), and also benefit from nutrient therapy to elevate folate levels. This approach is based on biochemical studies of 3,600 persons diagnosed with clinical depression, and is described in Chapter 6 of the new book Nutrient Power by Dr William Walsh.

Specific Recommendation

Doctors should perform blood testing prior to prescribing antidepressants, especially for young males. For example, inexpensive testing for whole-blood histamine and blood serum folate levels can efficiently identify persons at risk for suicidal ideation and violence. 

William J. Walsh, PhD
Walsh Research Institute, Naperville, IL

Robert A. deVito, MD              
Professor Emeritus, Loyola University –  Stritch School of Medicine

[NOTE:   For the full list of 4,800+ media articles naming antidepressants, see ssristories.com ]

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General